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A group of endogenous Cushing syndrome (CS) that may result from excess secretion of cortisol by either a unilateral and benign (adrenocortical adenoma), or malignant (adrenocortical carcinoma) adrenocortical tumor, or nodular adrenocortical disease, including Cushing syndrome due to bilateral macronodular adrenocortical disease (BMAD), isolated micronodular adrenocortical disease (i-MAD) and primary pigmented nodular adrenocortical disease (PPNAD; isolated or in Carney complex).
No hypotheses generated for this disease yet.
The autonomous pipeline will investigate this disease when evidence emerges.